How PEMF Therapy Helps With Weight Loss & Obesity Studies.

HOW “PEMFs” AID IN OBESITY MANAGEMENT


PEMFs can contribute to obesity management in several ways:
– Reducing inflammation by lowering the levels of proinflammatory cytokines.
– Increasing adiponectin levels while decreasing leptin levels.
– Reducing the production of fat.
– Breaking down fat cells.
– Influencing appetite.

Reducing Inflammation by Lowering Proinflammatory Cytokine Levels.
PEMFs reduce inflammation by lowering the levels of several proinflammatory tissue cytokines, including tumor necrosis factor-α (TNF-α), interleukin (IL)-1β, IL-6, and IL-8 (Vincenzi, 2017), as well as NF-kappaB (Vianale).

However, very low-intensity PEMFs, between 3 and 5 microTesla, do not affect some of these cytokines (de Kleijn). While these cytokines are mentioned for informational purposes, it is important to note that PEMFs can affect them in various health contexts beyond obesity, indicating their broad anti-inflammatory actions.

Raising Adiponectin Levels and Lowering Leptin Levels.
Stem cells derived from fatty tissue, known as adipose-derived stem cells (ADSCs), are frequently used in stem cell therapies. These cells contribute to the formation of new fat cells and release cytokines as they grow. A study conducted on rats examined the impact of PEMFs on the production of adiponectin and leptin by ADSCs.

Fatty tissue was harvested from the rats, and the stem cells were isolated and cultured. PEMF stimulation (7 Hz at 30 mT/300 Gauss) was applied to the cell cultures starting 24 hours after incubation, for 4 hours a day over 3 consecutive days.

The results showed that PEMF application to ADSCs from obese adult male rats increased adiponectin levels, while ADSCs from females exhibited lower leptin levels (Baranowska). Both outcomes are desirable, as they indicate an increase in adiponectin and a decrease in leptin levels.

Decreasing Fat Production.
Certain PEMFs may prevent stem cells in soft tissues (in cartilage, bone, bone marrow, or fat) from becoming fat cells. In another study, 7.5 Hz PEMFs at 4000 Gauss were used for 2 hours a day for 15 days in mice [Du]. Extremely low frequency PEMFs helped prevent stem cells from turning into fat cells, called fat cell differentiation.

Differentiation is when another type of cell acquires features of another type of cell. But, the PEMFs had no effect on the growth and function of the stem cells in making other types of cells. This suggests that stimulation of even undifferentiated soft tissue stem cells—not only in the abdomen but also in other parts of the body—may decrease the production of fat cells.

Breaking Down Fat Cells.
In another study, a 2-Gauss PEMF was combined with micropressure in 28 women and men over 6 weeks (Beilin). There was a reduction in waistline of more than 6 centimeters (about 2 and half inches) after 12 sessions. Researchers assumed this was due to a reduction in the size of fat cells.

Affecting Appetite.
A study was done on obese and nonobese females to see if PEMF stimulation to the brain could impact obesity by reducing appetite. A medium intensity of 45 mT PEMF with alpha range frequencies was applied across the head near the temples for 10 to 15 daily sessions.

Six months after treatment, the study group showed an average reduction in body mass index by 5.9 kg/m2 and the nonobese group by 2.5 kg/m2. Lipids returned to normal in 70% of the participants. In another study by the same group, 84 adolescent girls saw an average reduction of body weight by 9 kilograms (almost 20 pounds) after 3 months (Bolotova).

Conclusion.
Obesity is a challenge not only in terms of the causes but also in the consequences. Beyond the well-known consequences of obesity—hypertension, heart disease, gallbladder disease, arthritis, sleep apnea—less known is the major importance of inflammation associated with obesity not only in the obese tissues themselves but also throughout the body. This inflammation is associated with increased production of inflammatory cells—cytokines/adipokines, chemokines, and autoimmune complexes—in the fatty tissue itself and throughout the body.

PEMFs have been shown to reduce inflammation in many conditions through numerous mechanisms, including the inflammation associated with obesity. Since the visceral/abdominal fat compartment is the greatest source of inflammation with obesity, application of PEMFs to the abdomen covering a wide enough area with a sufficient intensity applied daily to anybody of any level of obesity is expected to significantly help with existing inflammation and with preventing the damaging consequences of the secondary inflammation throughout the body. A better option would be to apply sufficient intensity whole-body PEMFs to help not only visceral, abdominal, fat and the fat under the skin but also the systemic effects of the inflammation caused by any excess fat.

 

Reference:

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